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Chronic Lyme disease: Persistent musculoskeletal and neurocognitive symptoms with fatigue occur in a small percentage of pts after antibiotic treatment. Further antibiotic courses are not helpful; treatment consists of symptom-based supportive care. Although convalescence is longer the later antibiotics are given, the overall prognosis remains excellent, with minimal or no residual deficits. Clinical Features Disease is manifest by primary skin lesions that become disseminated with time. Diagnosis Diagnosis is based on clinical presentation and dark-field microscopy of scrapings from lesions. Rodents, particularly rats, are the most important disease reservoir, but many mammalian species can harbor the organisms. Approximately 40?20 cases are reported each year in the United States, but these numbers are likely to represent significant underestimates. Risk factors in the United States include recreational water activities, occupational activities that result in exposure to animals or animal waste. Pathogenesis Entry of the organisms via skin abrasions or intact mucous membranes is followed by leptospiremia and widespread dissemination. Clinical Features ?Incubation period, 1? weeks (range, 2?0 days) ?Anicteric leptospirosis, a biphasic illness, is the milder form and is found in 90% of symptomatic cases. Symptoms subside within 1 week and recur after 1? days in conjunction with antibody development. Symptoms are generally milder in phase 2, but up to 15% of pts can develop clinically evident aseptic meningitis. After 4? days of mild illness, more severe symptoms develop; however, illness is not truly biphasic. Hemorrhagic manifestations commonly include epistaxis, petechiae, purpura, and ecchymoses. Milder cases can be treated with oral doxycycline (100 mg bid) or amoxicillin (500 mg qid). About 35 cases per year are reported in the United States, mostly in forested mountainous areas of far western states and among persons sleeping in rustic mountain cabins and vacation homes. Sudden onset of high fever, headache, shaking chills, sweats, dizziness, nausea, vomiting, myalgias, arthralgias (sometimes severe); no arthritis 3. Each episode is less severe and is followed by a longer afebrile interval than the last. In the United States, the prevalence is highest in the south-central and southeastern states. Pathogenesis Rickettsiae are inoculated by the tick after 6 h of feeding, spread lymphohematogenously, and infect numerous foci of contiguous infected endothelial cells. Macules typically appear on the wrists and ankles, subsequently spreading to the rest of the extremities and the trunk. Such petechiae eventually develop in 41?9% of pts, appearing on or after day 6 of illness in ~74% of all cases that include a rash. The palms and soles become involved after day 5 in 43% of pts but do not become involved at all in 18?4%. Pts develop hypovolemia, prerenal azotemia, hypotension, noncardiogenic pulmonary edema, and cardiac involvement with dysrhythmias. Pulmonary disease is an important factor in fatal cases and develops in 17% of cases overall. Renal and hepatic injury can occur, and bleeding is a rare but potentially life-threatening consequence of severe vascular damage. Other laboratory findings may include increased plasma levels of acute-phase reactants such as C-reactive protein, hyponatremia, and elevated levels of creatine kinase. Prognosis Without treatment, the pt usually dies in 8?5 days; a rare fulminant presentation can result in death within 5 days. The mortality rate was 20?25% in the preantibiotic era and remains at 3?% despite the availability of effective antibiotics, mostly because of delayed diagnosis. Disease is characterized by high fever, rash, and-in most locales-an inoculation eschar (tache noire) at the site of the tick bite. A severe form of disease with ~50% mortality occurs in pts with diabetes, alcoholism, or heart failure. Doxycycline (100 mg bid for 1? days), ciprofloxacin (750 mg bid for 5 days), or chloramphenicol (500 mg qid for 7?0 days) is effective for treatment.

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Comparison of fracture neck femur and trochanteric fracture Features Age Incidence Blood loss Mechanism Signs: Shortening Deformity Site of tenderness Conservative treatment Surgery Complications Nonunion Malunion Fracture neck femur Elderly Common Less Trivial fall Minimum Minimum external rotation Anterior hip joint line Not successful Absolutely indicated Very common Unheard Trochanteric fracture More elderly Four times more common More Major trauma Gross Gross external rotation Over greater trochanter Successful Indicated for early mobilization Rare (1%) Very common Fig. Coxa vara, nonunion is less than 2 percent (rare) and traumatic osteoarthritis is seen. Drugs: Whose prolonged use causes osteoporosis are heparin, methotrexate, ethanol, glucocorticoids, etc. Chronic illness like rheumatoid arthritis, cirrhosis, sarcoidosis, renal tubular acidosis, etc. Endocrine abnormalities: Hyperparathyroidism, increased levels of glucocorticoids, estrogens, etc. Remember In osteoporosis ?Decreased density is due to deficiency of protein matrix in which calcium is laid down. Definition It is a generic term referring to a state of decreased mass per unit volume of a normally mineralized bone due to loss of bone proteins. It is called as silent epidemic and usually remains undetected till the patient sustains a hip, rib or spine fracture. Remember About osteoporosis It is the most common skeletal disorder in the world, next only to arthritis. Most prevalent complications are fractures of vertebral bodies, ribs, proximal femur, humerus, distal radius with minimal trauma. Criteria for screening: the following group of people need to be screened: ?All women > 65 years of age ?All men > 70 years of age ?Selected post-menopausal men and women who are 50-69 years with risk factors for fractures. Clinical Features Early symptoms: the patient complains of acute pain in middle or low thoracic or high lumbar region (Fig. Most common symptom of osteoporosis is back pain secondary to vertebral compression. However, in some cases, fractures of axial skeleton may be seen with trivial trauma. Round type of gibbus due to compression of thoracic vertebrae is commonly seen (Fig. Investigation Radiographs Radiographs changes seen in the spine are: ?Loss of vertebral height due to symmetric transverse compression. Transiliac bone biopsy: It is an important diagnostic tool in patients of more than 50 years in postmenopausal diseases. Blood chemistry: Serum calcium, phosphorus and alkaline phosphatase levels are usually normal. Management of Osteoporosis Preventing osteoporosis is lot easier than treating it. Drug Therapy in Osteoporosis Drugs form the mainstay of treatment of osteoporosis. However, an effort is made here to provide a simplistic analysis of the drugs commonly used in osteoporosis. Hormone Replacement Therapy the role of estrogen and progestogens in preventing and treating osteoporosis has been well documented. Biphosphonates these drugs inhibit the action of the osteoclast bone cells, which are responsible for removing the bone mass by binding themselves to the inner linings of the bones. It is used for the treatment of osteoporosis in women who are at least five years postmenopausal and in some cases of men. It is known to slow down bone loss, as it is a powerful inhibitor of osteoclastic activity, increase bone density and reduce the risk of fractures. Alfacalcidol this is a synthetic analogue of calcitriol, an active metabolite of vitamin D. Role of Fluorides in the Treatment of Osteoporosis Fluorides are known to increase the bone mass. Lower dose of 25 mg slow release fluorides twice daily along with 400 mg of calcium twice daily is recommended. The side effects are gastrointestinal upsets and increased risk of cortical bone fractures. Though the initial choice of the drugs depends on various factors like sex, age, presence or absence of uterus in women, tolerability, etc. An effort is made here to present the more appropriate of the drug options in the order of preference. Common Preference Calcium supplements in the dose of 1000-1500 mg/ day and vitamin D analogue (0.

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K-wire fixation is considered in patients like dentist or surgeon who wants to return to work quickly. Jersey finger: It is due to avulsion of flexor digitorum profundus from its insertion on distal phalanx. This consists of extensive metaphyseal comminution with involvement of the entire articular surfaces and bone loss. Clinical Features Pain, swelling, tenderness, deformity of the finger and loss of finger functions are the usual complaints. Hand Injuries 199 volar base fractures with less than 40 percent involvement of the articular surface. Closed reduction and internal fixation: this is indicated for both dorsal and volar base fractures of the middle phalanx with less than 40 percent articular surface involvement (Figs 16. Dynamic traction: this is a unique method of treatment and is indicated in volar base fractures of more than 40 percent and in the very difficult Pilon fractures. Volar plate arthroplasty: this is indicated in chronic injuries and in volar base fractures greater than 40 percent. Open reduction and internal fixations: this is indicated in single large fragment and in fixing bone graft to the metaphysis. For dorsal base fractures, extension block pinning after closed reduction is the treatment of choice. Stress tests: Lateral stress testing is performed with the fingers in complete extension and 30?of flexion. Operative Management Open reduction is indicated for open injuries, irreducible dislocations and injury to the collateral ligament of the index finger. Salient Features ?Due to the deforming forces of the intrinsic muscles, transverse and short oblique fractures of the proximal phalanx angulate dorsally. Treatment Methods Nonoperative Treatment this is indicated for undisplaced and for reducible but stable extra-articular fractures. The options for internal fixation after open reduction are: ?Intraosseous wiring. Pathognomonic sign is the presence of sesamoid bones within the joint and puckering of the volar skin. Treatment Nonoperative Treatment this is indicated in simple dorsal dislocations and collateral ligament ruptures. Operative Treatment this is indicated in complex dorsal dislocations, volar dislocation and radial collateral ligament injury of the index finger. The procedure consists of open reduction followed by repair or reconstruction of the collateral ligaments. Here there is an interposition of volar plate between the base of the proximal phalanx and the head of the metacarpal (Fig. Incidence: this is commonly seen in the index finger next is thumb, little finger. Both results from hyperextension injuries and in both the volar plate are torn at its proximal insertion into the metacarpal neck. These fractures should be accurately reduced with no rotational malalignment and immobilized with either plaster (common) or percutaneous or open K-wire fixation (less common). Clinical Features Pain, swelling, tenderness over the dorsum of the hand and loss of the hand functions are the usual complaints (Figs 16. Treatment Methods Nonoperative Treatment: this is indicated in the following situations: ?Undisplaced fractures. Methods: the hand can be immobilized by: ?Burk halter splint: this is ideal and is known to give good splints. Closed Reduction and Internal Fixation this is indicated for fractures that are unstable after reduction and for base fractures. This is mainly used for extra-articular fractures but can also be used for intra-articular fractures that are stable with K-wire fixation alone after reduction. Open Reduction and Internal Fixation this is indicated in the following: ?Multiple fractures ?Open fractures ?Irreducible fractures ?Displaced intra-articular fractures. The choice of fixation should be the one with which the surgeon is most familiar with (Figs 16.

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Infections often involve multiple species of anaerobes combined with microaerophilic and facultative bacteria. Major anaerobic gram-positive rods include spore-forming clostridia and non-spore-forming Propionibacterium acnes (a rare cause of foreign-body infections). Pts have sore throat, foul breath, fever, a choking sensation, and tonsillar pillars that are swollen, red, ulcerated, and covered with a gray membrane. This condition initially represents a chemical injury and not an infection, and antibiotics should be withheld unless bacterial infection supervenes. Pts have symptoms resembling other anaerobic pulmonary infections but may report pleuritic chest pain and marked chest-wall tenderness. Pure anaerobic infections occur more often at pelvic sites than at other intraabdominal sites. Pts may have foul-smelling drainage or pus from the uterus, generalized uterine or local pelvic tenderness, and fever. Diagnosis When infections develop in close proximity to mucosal surfaces normally harboring anaerobic flora, the involvement of anaerobes should be considered. The three critical steps in successfully culturing anaerobic bacteria from clinical samples are (1) proper specimen collection, with avoidance of contamination by normal flora; (2) rapid specimen transport to the microbiology laboratory in anaerobic transport media; and (3) proper specimen handling. Aerobic gram-negative flora should also be treated, with coverage for enterococci when indicated. Epidemiology In the United States, ~1100 cases of nocardial infection occur annually, of which 85% are pulmonary or systemic. Pathology and Pathogenesis Pneumonia and disseminated disease follow inhalation of bacterial mycelia. Clinical Features ?Pulmonary disease is usually subacute, presenting over days to weeks. Extrapulmonary disease is documented in >50% of cases, and some pulmonary involvement is evident in 80% of pts with extrapulmonary disease. A prominent cough productive of small amounts of thick purulent sputum, fever, anorexia, weight loss, and malaise are common; dyspnea, hemoptysis, and pleuritic chest pain are less common. Cellulitis: Subacute cellulitis may present 1? weeks after a break in the skin (often contaminated with soil). The firm, tender, erythematous, warm, and nonfluctuant lesions may involve underlying structures. Actinomycetoma: A nodular swelling forms at the site of local trauma, typically on the feet or hands. Fistulae form and discharge serous or purulent drainage that can contain granules consisting of masses of mycelia. Nocardiosis Table 100-1 lists the drugs, dosages, and durations used for treatment of nocardiosis. For serious disease, serum sulfonamide levels should be monitored and maintained at 100?50 g/mL. Once disease is controlled, the trimethoprim-sulfamethoxazole dose may be decreased by 50%. Amikacin drops Drugs for systemic therapy as listed above Topical: Until apparent cure Systemic: Until 2? mo after apparent cure each category, choices are numbered in order of preference. This diagnosis should be considered when a chronic progressive process with mass-like features crosses tissue boundaries, a sinus tract develops, and/or the pt has evidence of a refractory or relapsing infection despite short courses of antibiotics. Most infections are polymicrobial, but the role of other species in the pathogenesis of the disease is unclear. Its incidence is decreasing, probably as a result of better dental hygiene and earlier initiation of antibiotic treatment. Local infection spreads contiguously in a slow, progressive manner, ignoring tissue planes. Lesions cross fissures or pleura and may involve the mediastinum, contiguous bone, or the chest wall. The disease usually presents as an abscess, mass, or lesion fixed to underlying tissue and is often mistaken for cancer.

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Rules under which questions will be answered: Only three questions can be submitted to be answered. Only one side of sheet to be written on; matter must be typewritten or else written 2. If a quick answer is desired by mail, a nominal charge of 25 cents research work oF Intricate calculations a special rate will be charged. This question cannot be answered exactly, excepting on one condition, and that is that you would need a magnetomometer at hand with which to measure precisely the strength of the magnetic flux in maxwells per square inch of pole-face area. Of course, the voltage can always be increased by raising the speed, but naturally there is a limit to this, as the machine will not safely stand too high a speed. This be 24 pies or coils, each secondary winding should give 14,176 volts with 300 turns in the primary; 21,265 volts on 200 turns in the primary, and 42,530 volts with 100 turns in the primary in use. Of course, when the normal number of primary turns are reduced in order to raise the secondary voltage, a suitable iron core % Q. We the primary winding of iiiiiliiuii this transformer We consists of 13}4 pounds or 244 turns of No. Also in the case of the solenoid, with a movable core, the current will vary from a very high value when the core is just entering the coil, down to a certain minimum value, when the core is pushed all the way into the coil. The only thing we can suggest in your case would be to experiment with several For the size of solenoid you sizes of wire. Close-Ups of the Newest Scientific Movies the Role of Electricity and amperage as possible - from a dynamo as Science in Photoplays. The weight which such a solenoid could lift with the core all the way in the coil would probably be in the neighborhood of oneThe iron core must be lamhalf pound. The armature has 28 slots, wound with 28 coils, parallel lap winding, and has 2 brushes at an angle of 60 degrees from each other. Commutator has 28 bars, diameter is 4^4 inches, length 3 inches, and at a speed of 2,000 R. The dynamo is shunt wound, and when wired for 40 volts I want It seems to pull very hard it shunt also. We would advise as follows concerning the rewinding of your 105 volt, 9 follows Field - K. Johnson, McConnelsville, Ohio, writes this department: Please give data for winding a Q. The laminated sheet iron core should measure 15 inches long and 8}4 inches wide, and have a cross-section of 2 X 2 inches, or 4 square inches. The primary winding which should have taps taken off from each layer should comprise 344 turns of No. The armature should be rewound with the same number of coils and in the same fashion or style of winding as previously used, but each coil should be composed of 7 turns of No. You mention that the dynamo armature seems to turn very hard without any load on it at the present time. This most likely is not due to any electrical reasons, but merely mechanical ones. We would suggest that the bearings be carefully inspected; also on some small dynamos fitted with a number of brushes, the brush friction is made too great, and they should be readjusted so as to have just sufficient spring tension against the commutator to elimIn some cases, inate any undue sparking. Even draftsmen of limited training and experience are snapped up and paid good salaries. And now when American industries are to be called upon to meet vast foreign and increased domestic demands, the opportunities are gTeater than ever. Come to the College or Learn At Home e^C2>0 Hold you present position while training. Easy Payments fees for Chicago "Tech" Courses are very moderate-and you can pay on easy terms. And also, you obtain in a few months what it would take several years to acquire by ordinary methods. You are soon ready to take a paying position and to quickly get back the coat of your course. These instruments are of the same make and sizes as are used by high salaried experts in drafting rooms of factories, shops, railroads, etc. Send the Coupon the coupon Learn Ail this in Spare Time Principles of the Automobile, Every point made clear about the baai Dapm In pleasure and commercial cars, All about the different types.

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Necrosis is most pronounced in the front and causes the vertebral body to become wedge shaped T6 (Figure 5. The normal curvature of the T7 are congenital changes, fractures, growth disturbance. Sometimes it is difficult to distinguish between kyphosis and a normal relaxed posture. Most patients with moderate changes in the thoracic spine (which have been identified by X-ray) are pain free. Patients with Scheuermann disease in the thoracolumbar or the lumbar region usually have pain. If the diagnosis is suspected during the growth period, the athlete should be referred for appropriate orthopedic care. T8 T9 T10 T11 Morbus Bekhterev-Bekhterev Disease Bekhterev disease occurs more often than Scheuermann disease and makes its first appearance earlier in men than in women. In Norway, the prevalence is about 1? per 1000 in the adult population, with 300?00 new cases annually. The condition is hereditary, and it is assumed that transmission is multifactorial. Lateral X-ray shows an athlete with increased thoracic kyphosis, due to a wedge shape of the vertebral bodies of more than 5?with permission from the Norwegian Sports Medicine Association. A diagnosis is made if reduced range of motion in one or more sections of the back is present, as well as tenderness to palpation of the spinous processes and the iliosacral joints. Bekhterev disease affects the ischial tuberosity, the iliac crest, and the tendon insertions in the heel. It may make its first appearance with recurring eye inflammation (iridocyclitis), and it affects large joints (shoulders and hips). Chest excursion may be reduced early, but major limitations are usually present during the late stages. A radiographic examination of the pelvis and vertebral column are diagnostic but rarely during the onset stage. Patients with severe pain should be evaluated by a rheumatologist for the use of medication. Surgical treatment may be indicated if the patient has major thoracic kyphosis that makes it impossible to maintain a normal visual angle. The frontal X-ray view demonstrates significant sclerosis as an of the longitudinal ligaments. Rehabilitation starts at the time of the acute injury and comprises the period of acute care, sport-specific training and return to competition. A thorough psychosocial evaluation to identify barriers to improvement is often helpful and it is recommended to include this early in the rehabilitation process. The acute phase may be dramatic and the evaluation should include a biomechanical and psychological evaluation in order to gain confidence and reduce fear. Imaging may be important in the acute or subacute phase, but the indication and interpretation should be evidence based. For example, abnormal findings may be normal according to age and not explain the pain and disability observed. The challenge is to promote confidence and understanding of the physical injury as well as the emotional reaction involved. Goals Acute phase Create calm and confidence, improve pain management, limit inflammation, invalidate red flags Create a rehabilitation plan Restore normal function Maintain general strength and endurance Measures sion, and elevation) Create an overview of the diagnosis, prognostic factors, and sports activity, and create a plan in consultation with the athlete and trainer Advice based on the anticipated natural course Rehabilitation phase In case of chronic pain, chart yellow flags and counteract passive pain mastery Individually adjusted alternative training program Counteract the risk factors based on the sport-specific, individual, and general evaluation Training phase Lead the athlete back to sport activity dent evaluations of their career prospects Table 5. It is often difficult to become completely pain free using relief from usual activities and drug therapy. In the natural course of acute neck and back injuries, the athlete should recover well in a short time. How long training and competition are interrupted will depend on the condition of the athlete and the requirements of the sport. Training to maintain general endurance and strength can often begin within a few days of injury. Function often improves significantly during the first week and makes treatment and rehabilitation unnecessary.

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Remember the drugs used in the prevention and treatment of gout: ?Indomethacin ?Colchicines prevention ?Allopurinol ?Steroids. Surgery for rheumatoid arthritis; timing and techniques: general and medical aspects. Larger joints are more affected and 50 percent involve the knee joints unlike in gout (Fig. Causes In cerebral palsy, the causes are different in prenatal, natal, postnatal and perinatal period and are listed as in Table 42. Lesions In cerebral palsy, the lesion could be in either the brain or the upper cervical cord, and the lesion is static. Classification Cerebral palsy is classified based on various clinical types and based on the degree of severity. Lesions in the Brain In in ?cerebral palsy, the lesions in the brain can occur the following four areas: Cerebral cortex (spastic type) Midbrain (dyskinesia) Cerebellum (ataxic) Table 42. Other clinical features depend on the geographic distribution of cerebral palsy and the associated handicapping situations (Table 42. Orthopedic Deformities the following are the common orthopedic deformities encountered in cerebral palsy. Knee ?Genu recurvatum Genu valgum Patella alta Subluxation or dislocation of patella. The role of orthopedic surgeon starts when the child is 12 months of age and seldom before. Operation on muscles and tendons: ?Tenotomy, tendon lengthening and tendon transfers. Operation on bones and joints: ?Bone lengthening or bone shortening to equalize the limb lengths. Neuromuscular Disorders 603 ?Arthrodesis of wrist, hip and foot to correct deformity, provide stability and to improve functions. Pitfalls: It has no effect on shortened and contracted muscles and it is here that the orthopedic surgery helps. Quadriplegics and total body involvement will never walk but can be propped sitters. On examination, there could be mild neck stiffness and the child may find it difficult to move the affected limb (preparalytic). The lower limbs are more commonly affected and the paralysis could be partial or total (paralytic stage) (Figs 42. The paralysis of the muscles whether spinal (75%) or bulbar (25%) usually lasts until two months. Any residual paralysis after two years of affection is permanent with no chance of recovery. Quick facts: Features of paralysis due to polio ?Lower limb is more commonly affected than the upper limbs. Pathogenesis the virus is transmitted through the feco-oral route, enters the nervous tissue, and destroys the anterior horn nerve cells because of which the peripheral nerve degenerates resulting in muscle and tendon atrophy (Fig. Orthopedic Deformities Orthopedic deformities encountered in poliomyelitis are listed in Table 42. Differential Diagnosis Poliomyelitis has to be differentiated in the acute stages from: ?Pyogenic meningitis ?Guillain-Barr?yndrome ?Postdiphtheritic paralysis Figs 42. The child is put on a ventilator support if there is respiratory paralysis due to bulbar polio. Warm and moist packs are given to the joints and all intramuscular injections are avoided during this phase. Recovery stages: In this stage, the joints are properly splinted through various appliances to prevent or correct the deformities (Table 42. Quick facts: Conservative treatment Stage of onset Stage of greatest paralysis Stage of recovery Stage of residual paralysis ?Bed rest Splints Artificial respiration, etc. Can be corrected by provision of suitable ?Orthotic appliances or by operation Knee Hip ?Flexion abduction contractures of the hip ?Paralysis of gluteus medius, maximus ?Paralytic dislocation of hip ?Lumbar scoliosis Pelvic obliquity Hip flexed and abducted External rotation of femur Flexion and valgus of knee Posterior and lateral subluxation of tibia External rotation of tibia Foot in equinus Shortening Iliotibial band contractures (Results in 9 classical deformities) Spine ?Kyphosis ?Scoliosis ?Kyphoscoliosis ?Paralysis of shoulder, elbow, forearm and hand muscles. Role of Appliances the purpose of external appliances is to support joints that have lost their normal control (Fig. In ?the late stages from: Cerebral palsy Spina bifida Myopathies Muscular dystrophies, etc.

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Diagnosis: Diagnosis is based on soreness to palpation over the pulley and to flexion motion in the affected finger. If several pulleys are torn, the flexor tendon may be palpated as a bowstring in the subcutaneous tissue. A trained radiologist should be able to see the ruptured pulleys by performing an ultrasound examination. Treatment: If a bowstring is palpated, the patient should be referred to surgery for suturing or reconstruction of the pulleys. The patient should be informed about what causes the condition and should use caution when returning to rock climbing after symptoms have subsided. When the pulleys are reconstructed, adhesions may develop that might reduce the flexion of the affected finger. The patient should wait 8?0 weeks after surgical intervention before resuming sport activity. Dislocations of the little finger or thumb are the most common injuries, but dislocation of any joint on any finger is reported. Symptoms and signs: Symptoms are swelling, tenderness, deformity, and limited range of motion. Diagnosis: Diagnosis is based on the presence of malalignment and instability, or history of the same. Radiographic examination is necessary to see if there are any associated fractures. Treatment: Reduction of a dislocated finger by pulling the finger in axial direction is usually easy to do immediately after the injury has occurred. A radiographic examination should be performed to confirm the reduction and to rule out any fracture while relocating the joint. The finger is immobilized or fixed to an adjacent finger with a cast or tape for 3? weeks, after which exercises may begin. The injured finger should be buddy taped to an adjacent finger for a few more weeks during sport activity. In cases of a fracture dislocation the athlete should be referred to a hand surgeon for evaluation of surgical treatment. Sport activity may be resumed 5? weeks after the injury occurs, but the finger should be protected by a buddy tape. A compression of a nerve may cause reduced sensation, but the continuity is intact. Dorsal finger nerves (primarily from the radial nerve) are usually not repaired, as this injury causes minor discomfort for the patient. However, an injured digital nerve on the palmar side of the finger should be referred for surgical treatment as soon as the injury is diagnosed, or no later than 1? weeks after injury. The patient may indicate whether sensation is any different on different sides of each finger when the radial and ulnar sides of each finger are examined as previously described. A combination of several measures is needed to rehabilitate major hand and finger injuries. It is preferable for an experienced hand therapist to administer or direct this therapy. A well-functioning hand must have motion, stability, strength, and sensation, and must be pain free. In cases in which stabilizing surgery is necessary, systematic conservative rehabilitation must follow, with the goal of achieving good long-term results. During the acute phase, it is important to gain control over pain and inflammation by cooling with ice, elevation, compression, and rest from activities that cause pain. During the rehabilitation phase, braces, splints, or taping to an adjacent finger are often used to ease resumption of active assisted mobilization of joints with the greatest possible joint motion. This can be accomplished by having the patient wear fitted compression gloves, individually adjusted finger stockings (or Coban), or Tubigrip on his hand or wrist. The hand or fingers should be moved around for 45 seconds in the hot water and then for 15 seconds in the cold water. If pain increases during activity, the activity must be stopped immediately and ice applied locally to the painful area for 15?0 minutes. To avoid overloading the tendons, isometric or concentric exercises are recommended for strength training during the initial training phase. Toward the end of the training phase, the exercises are increased to eccentric loading, so that the athlete is ready to meet the requirements of the sport.

References:

  • https://www.crohnscolitisfoundation.org/sites/default/files/legacy/assets/pdfs/ibd-and-irritable-bowel.pdf
  • http://www.nfid.org/publications/reports/rsv-report.pdf
  • https://www.brown.edu/Courses/Bio_281-cardio/cardio/EKGhandout2.pdf
  • http://158.232.12.119/entity/biologicals/vaccines/TRS_978_Annex_3.pdf
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